Advanced Search

Submit Online

Volume 11, Issue 3, March 2019

Editorial

p53: updates on mechanisms, biology and therapy (I) Free
Chandra S. Verma
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 185-186, https://doi.org/10.1093/jmcb/mjz017
Abstract | Full Text 

Article

A single synonymous mutation determines the phosphorylation and stability of the nascent protein Free
Konstantinos Karakostis , Sivakumar Vadivel Gnanasundram , Ignacio Lo´pez , Aikaterini Thermou, Lixiao Wang, Karin Nylander , Vanesa Olivares-Illana , and Robin Fa°hraeus
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 187-199, https://doi.org/10.1093/jmcb/mjy049
Abstract | Full Text 

Reviews

The role of p53 in developmental syndromes
Margot E. Bowen and Laura D. Attardi
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 200-211, https://doi.org/10.1093/jmcb/mjy087
Abstract | Full Text 
Discussion of some ‘knowns’ and some ‘unknowns’ about the tumour suppressor p53
Elizabeth Lieschke, Zilu Wang , Gemma L Kelly , and Andreas Strasser
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 213-223, https://doi.org/10.1093/jmcb/mjy077
Abstract | Full Text 
Functional relationship between p53 and RUNX proteins
Suk-Chul Bae, Arun Mouli Kolinjivadi , and Yoshiaki Ito
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 224-230, https://doi.org/10.1093/jmcb/mjy076
Abstract | Full Text 
The long and the short of it: the MDM4 tail so far
Sue Haupt, Javier Octavio Mejı´a-Herna´ndez , Reshma Vijayakumaran , Simon P. Keam ,and Ygal Haupt
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 231-244, https://doi.org/10.1093/jmcb/mjz007
Abstract | Full Text 
Small molecule activators of the p53 response
Marcus J. G. W. Ladds and Sonia Laı´n
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 245-254, https://doi.org/10.1093/jmcb/mjz006
Abstract | Full Text 
Drugging in the absence of p53
Obed Akwasi Aning and Chit Fang Cheok
J Mol Cell Biol, Volume 11, Issue 3, March 2019, 255-264, https://doi.org/10.1093/jmcb/mjz012
Abstract | Full Text 

< Previous

Next >

Cover: The tumour suppressor p53 can stimulate a number of different cellular processes after it is activated by stress signals. p53 has multi-layered control over these diverse cellular responses, by directly (bold) or indirectly (italics) causing activation (in black) or repression (in purple) of relevant target genes; a subset of which are shown here. These processes are thought to contribute to the ability of p53 to suppress tumour development. See pages 212–223 by Lieschke et al. for details.