Volume 12, Issue 12, December 2020

Editorial

SARS-CoV-2 and its infected world
Jiarui Wu
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 913-913, https://doi.org/10.1093/jmcb/mjaa078
Abstract | Full Text 

Research Highlight

A promiscuous interaction of SARS-CoV-2 with bacterial products
Guillaume Carissimo, Lisa F.P. Ng
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 914-915, https://doi.org/10.1093/jmcb/mjaa068
Abstract | Full Text 

Articles

SARS-CoV-2 spike protein binds to bacterial lipopolysaccharide and boosts proinflammatory activity
Ganna Petruk, Manoj Puthia, Jitka Petrlova, Firdaus Samsudin, Ann-Charlotte Stro¨mdahl, Samuel Cerps, Lena Uller, Sven Kjellstro¨m, Peter J. Bond, Artur Schmidtchen
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 916-932, https://doi.org/10.1093/jmcb/mjaa067
Abstract | Full Text 
Histone deacetylase 4 inhibits NF-κB activation by facilitating IκBα sumoylation
Qi Yang, Jielin Tang, Chonghui Xu, He Zhao, Yuan Zhou, Yanyi Wang, Min Yang, Xinwen Chen, Jizheng Chen
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 933-945, https://doi.org/10.1093/jmcb/mjaa043
Abstract | Full Text 
Proteome-wide data analysis reveals tissue-specific network associated with SARS-CoV-2 infection 
Li Feng, Yuan-Yuan Yin, Cong-Hui Liu, Ke-Ren Xu, Qing-Run Li, Jia-Rui Wu, Rong Zeng
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 946-957, https://doi.org/10.1093/jmcb/mjaa033
Abstract | Full Text 

Perspective

COVID-19 and diabetes: the contributions of hyperglycemia
Jing Wang, Wen Meng
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 958-962, https://doi.org/10.1093/jmcb/mjaa054
Abstract | Full Text 
SARS-CoV-2 infection and the antiviral innate immune response
Hui Yang, Yingying Lyu, Fajian Hou
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 963-967, https://doi.org/10.1093/jmcb/mjaa071
Abstract | Full Text 

Reviews

Cytoskeleton—a crucial key in host cell for coronavirus infection
Zeyu Wen, Yue Zhang, Zhekai Lin, Kun Shi, Yaming Jiu
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 968-979, https://doi.org/10.1093/jmcb/mjaa042
Abstract | Full Text 
The development of neutralizing antibodies against SARS-CoV-2 and their common features
Liu Daisy Liu, Chaoyang Lian, Leng-Siew Yeap, Fei-Long Meng
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 980-986, https://doi.org/10.1093/jmcb/mjaa070
Abstract | Full Text 

Application Note

Rapid quantitative screening assay for SARS-CoV-2 neutralizing antibodies using HiBiT-tagged virus-like particles
Kei Miyakawa, Sundararaj Stanleyraj Jeremiah, Norihisa Ohtake, Satoko Matsunaga, Yutaro Yamaoka, Mayuko Nishi, Takeshi Morita, Ryo Saji, Mototsugu Nishii, Hirokazu Kimura, Hideki Hasegawa, Ichiro Takeuchi, Akihide Ryo
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 987-990, https://doi.org/10.1093/jmcb/mjaa047
Abstract | Full Text 

Corrigendum

Corrigendum to ‘New therapy targeting differential androgen receptor signaling in prostate cancer stem/progenitor vs. non-stem/progenitor cells’
Soo Ok Lee, Zhifang Ma, Chiuan-Ren Yeh, Jie Luo, Tzu-Hua Lin, Kuo-Pao Lai, Shinichi Yamashita, Liang Liang, Jing Tian, Lei Li, Qi Jiang, Chiung-Kuei Huang, Yuanjie Niu, Shuyuan Yeh, Chawnshang Chang
J Mol Cell Biol, Volume 12, Issue 12, December 2020, Pages 991-992, https://doi.org/10.1093/jmcb/mjaa077
Abstract | Full Text 
Cover: In Chinese ancient folklore, ‘Year’ ( ) is a ferocious monster, which brings disease and disaster to the world on New Year’s Eve. In early 2020, the monster ‘ ’ (SARS-CoV-2) spread worldwide causing a continued threat of the COVID-19 pandemic to the human society (village). The bridges in front of the village (hyperglycemia) may increase the susceptibility of SARS-CoV-2 infection and worse the prognosis, linking diabetes and COVID-19. Therefore, people set off firecrackers, beat gongs and drums, and paste spring couplets and paper cutouts (controlling blood glucose levels) to resist and drive away the monster ‘ ’. See pages 958–962 by Wang and Meng for details.